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2 edition of Lipoproteins and coronary atherosclerosis found in the catalog.

Lipoproteins and coronary atherosclerosis

International Symposium on Lipoproteins and Coronary Atherosclerosis (1981 Lugano, Switzerland)

Lipoproteins and coronary atherosclerosis

proceedings of the International Symposium on Lipoproteins and Coronary Atherosclerosis held in Lugano, Switzerland, October 1-3, 1981

by International Symposium on Lipoproteins and Coronary Atherosclerosis (1981 Lugano, Switzerland)

  • 356 Want to read
  • 35 Currently reading

Published by Elsevier Biomedical Press, Elsevier Science Pub. Co. [distributor] in Amsterdam, New York, New York .
Written in English

    Subjects:
  • Coronary heart disease -- Congresses.,
  • Blood lipoproteins -- Metabolism -- Congresses.,
  • Atherosclerosis -- Congresses.,
  • Lipoproteins -- Metabolism -- Congresses.,
  • Coronary disease -- Congresses.

  • Edition Notes

    Includes bibliographies and indexes.

    Statementeditors, G. Noseda ... [et al.].
    SeriesSymposia of the Giovanni Lorenzini Foundation ;, v. 13
    ContributionsNoseda, Giorgio.
    Classifications
    LC ClassificationsRC685.C6 I575 1981
    The Physical Object
    Paginationxi, 450 p. :
    Number of Pages450
    ID Numbers
    Open LibraryOL3482075M
    ISBN 100444804080
    LC Control Number82001450

    Atherosclerosis, the underlying cause of cardiovascular disease, is characterized by chronic inflammation and altered immune response. Cholesterol is a well-known risk factor associated with the development of cardiovascular diseases. Elevated serum cholesterol is unique because it can lead to development of atherosclerosis in animals and humans even in the absence of Cited by: The serum lipoprotein transport system in health, metabolic disorders, atherosclerosis and coronary heart disease by John W. Gofman, Oliver Delalla, Frank Glazier, Norman K. Freeman, Frank T. Lindgren, Alex V. Nichols, Beverly Strisower, Arthur R. TamplinFile Size: 1MB.


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Lipoproteins and coronary atherosclerosis by International Symposium on Lipoproteins and Coronary Atherosclerosis (1981 Lugano, Switzerland) Download PDF EPUB FB2

Arterial injury causes endothelial dysfunction promoting modification of apoB containing lipoproteins and infiltration of monocytes into the subendothelial space. Internalization of the apoB containing lipoproteins by macrophages promotes foam cell formation, which is the hallmark of the fatty streak phase of atherosclerosis.

Clean, undamaged book with no damage to pages and minimal wear to the cover. Spine still tight, in very good condition. Remember if you are not happy, you are covered by our % money back by: Atherosclerosis which accounts in Western Europe for more than 40 % of deaths, is a generalized disease that develops slowly and is symptomless until lesions have become sufficiently severe to cause myocardial or cerebral infarction.

Research on specific and precocious markers of atherosclerosis. The 51 contributions are organized into five parts.

Part I includes papers on the metabolism of lipoproteins. Part II presents studies on hormones and lipoproteins. Part III is devoted to atherosclerosis and lipoproteins.

Lipoproteins and Coronary Atherosclerosis: International Symposium Proceedings, 2nd Edition is a straightforward introduction to the core of programming. The Author dispenses with the sort of tedious, unnecessary information that can get in the way of learning how to program, choosing instead to provide a foundation in general concepts, fundamentals, and problem solving.

Three real world projects in the second part of the book. This chapter describes LDL, triglyceride‐rich lipoproteins (TRLs), and HDL, and their relationship to atherosclerosis.

Lipoproteins are important risk factors for atherosclerotic cardiovascular disease (CVD). HDL cholesterol (HDL‐C) is a major independent inverse risk factor for coronary heart disease (CHD).

Association of remnant-like particle cholesterol with coronary artery disease in patients with normal total cholesterol levels American Heart Journal, Vol.

No. 2 Plasma lipoproteins and progression of coronary artery disease evaluated by angiography and clinical events. Cooperative Study of Lipoproteins and Atherosclerosis. Evaluation of serum lipoprotein and cholesterol measurements as predictors of clinical complications of atherosclerosis: report of a cooperative study of lipoproteins and atherosclerosis.

() Circulation. Study Category: The Cohort Studies () Year Begun: The Role of Lipids and Lipoproteins in Atherosclerosis Author(s): John W. Gofman, Frank Lindgren, Harold Elliott, William Mantz, John Hewitt, Beverly Strisower, Virgil Herring and Thomas P.

Lyon Source: Science, New Series, Vol. No. (Feb. 17, ), pp. + Published by: American Association for the Advancement of ScienceFile Size: KB. Lipoproteins and coronary atherosclerosis book Analysis of plasma or serum total cholesterol, triglycerides, and lipoproteins is usually performed on blood specimens obtained by venipuncture in tubes containing EDTA.

Because of a larger content of water, plasma levels have been found to be Lipoproteins and coronary atherosclerosis book 3% lower than matched serum Cited by: About this book.

This innovative book focuses on HDL and its relationships to triglyceride-rich particles. As new therapies for HDL raising become available, a comprehensive understanding the role of HDL in Coronary Heart Disease is of great importance.

Atherosclerosis is the underlying cause of most acute coronary syndromes (ACS) such as myocardial infarction and unstable angina, which are major causes of mortality in the Western world.

Histological studies have demonstrated that ACS are triggered by disruption of vulnerable atherosclerotic plaques which results in luminal thrombosis, but. About this book. Introduction. Atherosclerosis which accounts in Western Europe for more than 40 % of deaths, is a generalized disease that develops slowly and is symptomless until lesions have become sufficiently severe to cause myocardial or cerebral infarction.

High Density Lipoproteins and Coronary Atherosclerosis A Strong Inverse Relation With The Largest Particles Is Confined to Normotriglyceridemic Patients Jan Johansson, Lars A. Carlson, Christian Landou, and Anders Hamsten The relations of high density lipoprotein (HDL) subclasses to severity and rate of progression.

High Density Lipoproteins, Dyslipidemia, and Coronary Heart Disease [Schaefer, Ernst J.] on *FREE* shipping on qualifying offers. High Density Lipoproteins, Dyslipidemia, and Coronary Heart DiseaseAuthor: Ernst J. Schaefer. The endothelium is a major participant in the pathophysiology of diseases, such as atherosclerosis, diabetes and hypertension, and these entities are responsible for the largest part of cardiovascular mortality and morbidly.

Atherosclerosis has an open access mirror journal Atherosclerosis: X, sharing the same aims and scope, editorial team, submission system and rigorous peer review. Atherosclerosis brings together, from all sources, papers concerned with investigation on atherosclerosis, its risk factors and clinical sclerosis covers basic and translational, clinical and.

Proposed timeline of coronary atherosclerosis and contribution of microRNA (miR) transcoronary gradients (TCGs) at each step. Interestingly, at most stages, miR, miR, and mi appear to be in abundance throughout the disease process, while very few miRs have reduced gradients (or are ‘retained’ in the circulation).

Lipoproteins, atherosclerosis, and coronary heart disease. Amsterdam ; New York: Elsevier/North-Holland Biomedical Press, (OCoLC) Document Type: Book: All Authors / Contributors: N E Miller; Barry Lewis. Two types of lipoproteins carry cholesterol to and from cells.

One is low-density lipoprotein, or LDL. The other is high-density lipoprotein, or HDL. The amount of each type of cholesterol in your blood can be measured by a blood test. LDL cholesterol is considered the “bad” cholesterol, because it contributes to fatty buildups in arteries.

The central role of lipoproteins and inflammation in atherosclerosis has been validated in many studies. Genome-wide association studies identified the first genetic loci.

Coronary atherosclerosis causes coronary artery disease. Complicated atherosclerotic plaque disrupts the blood flow in the coronary circulation.

Impaired blood flow causes a corrupted supply and demand of the oxygen and the metabolites in the heart. This results in a decrease in coronary arterial blood flow and a decrease in oxygen : Hakan Saçlı, İbrahim Kara, Mehmet Kaan Kırali.

Variability in atherogenic lipoproteins and coronary artery disease progression Fernando Rivero, Francisco Sánchez-Madrid, Variability in atherogenic lipoproteins and coronary artery disease progression, European Heart Journal, Volume 39 there was no evidence linking this biological marker with subclinical by: 1.

The Diabetes Atherosclerosis Intervention Study (DAIS) (Vakkilainen et al., ) showed that lipid-modifying treatment decreased the angiographic progression of coronary atherosclerosis in subjects with type 2 diabetes.

This effect was related in Cited by: 5. This chapter discusses a study to investigate the levels and lipid composition of the major lipoprotein classes in coronary and cerebral atherosclerotic : V. Orbetzova, L. Dakovska, M. Orbetzov, R.

Rashev, F. Elazarová, N. Yavorska, M. Anastassova. 1. Highlights of Current Research in Atherosclerosis.- 2.

Plasma Lipoproteins: Recent Developments.- 3. Arterial Endothelium and Its Potentials.- 4. The Coronary Drug Project - Findings with Regard to Estrogen, Dextrothyroxine, Clofibrate and Niacin.- 5.

The Place of the Aorto-Coronary By-Pass Surgery in the Treatment of Ischemic Heart Disease.- : G. Manning. Get this from a library. Lipoproteins and atherosclerosis. [Claude L Malmendier; P Alaupovic; Fondation de recherche sur l'athérosclérose de la communauté française de Belgique.

International Colloquium] -- This comprehensive work deals with the major health problem of high lipid levels in blood, a cause of serious heart and vascular disease.

In book: Atherosclerosis - Yesterday, Today and Tomorrow c. Hypertension: increases permeability of the vessel wall to lipoproteins and promotes re- Focus on Coronary Atherosclerosis. Hormones and Atherosclerosis covers the proceedings of the conference held in Brighton, Utah on MarchThe book focuses on the role of hormones in atherosclerosis, as well as cholesterol metabolism and Book Edition: 1.

High cholesterol levels lead to clogged arteries, which can ultimately lead to heart disease. WebMD explains how cholesterol levels - both good and bad. CiteScore: ℹ CiteScore: CiteScore measures the average citations received per document published in this title.

CiteScore values are based on citation counts in a given year (e.g. ) to documents published in three previous calendar years (e.g.

– 14), divided by the number of documents in these three previous years (e.g. – 14). We would like to show you a description here but the site won’t allow g: coronary atherosclerosis.

Effect of fenofibrate on progression of coronary-artery disease in type 2 diabetes: the Diabetes Atherosclerosis Intervention Study, Cited by: Lipid-lowering secondary-prevention trials of coronary artery disease (CAD) have implicated triglyceride-rich lipoproteins as the main determinants of angiographic progression after elevated LDL cholesterol levels have been lowered with therapy.

The. Atherosclerosis is the principal underlying cause of cardiovascular and cerebrovascular disease in people of the Western world. Cigarette smoking has been implicated in both the initiation and exacerbation of the atherosclerotic process.

lipoproteins, very-low-density lipoproteins, and chylomicron remnants. These lipoproteins likely are atherogenic by pene-trating the arterial wall, leading to accumulation of cholesterol in the intimal space, foam cell formation, and atherosclerosis 2–5 (Figure).

Triglycerides are primarily carried by remnants, and. Atherosclerosis development requires lipid-carrying particles (lipoproteins) to penetrate the arterial wall.

This is what allows deposits of lipids, cholesterol and other substances to form a plaque. Atherosclerosis can be a precursor to cardiovascular events, including CHD.

Puchois P, Kandoussi A, Fievet P, et al. Apolipoprotein A-I containing lipoproteins in coronary artery disease. Atherosclerosis ; Barter PJ, Nicholls S, Rye KA, et al. Antiinflammatory properties of HDL. Circ Res ; Mackey RH, Greenland P, Goff DC Jr, et al. High-density lipoprotein cholesterol and particle concentrations.

Associations Between Lipoproteins and the Progression of Coronary and Vein-Graft Atherosclerosis in a Controlled Trial With Gemfibrozil in Men With Low Baseline Levels of. Abstract Significance: Lipoproteins, such as low-density lipoprotein, play a causal role in the development of atherosclerosis and coronary disease.

Recent Advances: Lipoproteins can stimulate vascular production of reactive oxygen species, which act as important signaling molecules in the cardiovascular system contributing to the pathophysiology Author: HaghikiaArash, LandmesserUlf. In atherosclerosis, the arteries are narrowed when fatty deposits called plaques build up inside.

Plaques typically contain cholesterol from low-density lipoproteins (LDL), smooth-muscle cells and fibrous tissue, and sometimes calcium. As a plaque grows along the lining of an artery, it produces a rough area in the artery's normally smooth surface.HDL inhibits the development of atheroma and coronary artery disease by transporting excess tissue cholesterol to the liver, where it is converted into bile acids and excreted (figure 5) Lower levels of HDL have been correlated with an increased risk of atherosclerosis, the primary cause of cardiovascular disease.

The principal HDL pathway.Original Article from The New England Journal of Medicine — Statin Therapy, LDL Cholesterol, C-Reactive Protein, and Coronary Artery Disease.